Coconut Oil Seen as Viable Option to Prevent and Treat Alzheimer’s Disease

 Coconut oil in a container with spoon and 2 coconuts.

by Brian Shilhavy.Editor, Health Impact News

For more than a years now, we have actually been releasing research study and reviews from individuals who have actually had success in reversing Alzheimer’s Disease with coconut oil. The research study and reviews are released on , part of the Health Impact News network.

During this exact same period, we have actually seen the pharmaceutical market stop working over and over once again to establish a drug that works in dealing with Alzheimer’s Disease.

Researchers in Australia are now studying coconut oil, and its special structure of medium chain fats, as a method to deal with and avoid Alzheimer’s Disease.

Starting from the basis that Alzheimer’s Disease is a type of diabetes, described in the past as a “Type 3” kind of diabetes, or what the Australian scientists describe as “brain glucose hypometabolism,” the research study concentrates on offering an alternative kind of energy to the brain in the kind of “ketone bodies.”

The title of their research study is: Potential of coconut oil and medium chain triglycerides in the avoidance and treatment of Alzheimer’s illness, released in the March 2020 edition of the journal Mechanisms of Ageing and Development.

It is an extensive take a look at the existing literature on this subject, with numerous referrals.

Here is the abstract :


Alzheimer’s illness (ADVERTISEMENT) is the most typical type of dementia. Presently, there is no reliable medication for the avoidance or treatment of ADVERTISEMENT. This has actually resulted in the look for alternative healing techniques.

Coconut oil( CO) has a special fat structure that is abundant in medium chain fats( MCFA), a significant part of which straight reaches the liver through the portal vein, consequently bypassing the lymphatic system.

Given that brain glucose hypometabolism is a significant early trademark of ADVERTISEMENT, noticeable well prior to the start of signs, ketone bodies from MCFA metabolic process can possibly work as an alternative energy source to make up for absence of glucose utilisation in the brain.

Additionally, neuroprotective antioxidant homes of CO have actually been credited to its polyphenolic material. This evaluation goes over how the metabolic process of CO and MCFA might assist in compensating the glucose hypometabolism observed in the ADVERTISEMENT brain.

Furthermore, we provide the existing proof of the neuroprotective homes of CO on cognition, amyloid-β pathogenicity, swelling and oxidative tension.

The existing evaluation addresses the impact of CO/MCFA on other persistent conditions that are danger aspects for ADVERTISEMENT, and addresses existing spaces in the literature relating to using CO/MCFA as a prospective treatment for ADVERTISEMENT.

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